Below is a just released abstract that essentially finds that if you:
1. Memorize something.
2. Wait 6 days and try to recall it. (could probably be more or less days)
3. While you are recalling it , put your hand in ice water. (other mild pain stimulation might work as well, like a cloths-pin clamped on the body part of your choice.)
4. When you try to recall it on the following day, you should remember it better than the day before.
Perhaps, however, just adding a little pain when you are first learning it would work as well.
Why this might work:
Memories come out of storage when recalled, and need to be remembered again (reconsolidated, the technical term) or they will be lost. Stress enhances the chemical environment in the brain that fosters learning, so there is a better chance that what is encountered during pain will be learned, and learned immediately.
Makes sense too. If you meet someone new and she shakes your hand there is a lot less of a chance you will remember them the next week (or the next year for that matter) then if she slapped your face instead.
…robust memory performance was shown at testing when the CPS administration was concurrent with the retrieved-labile memory state. Results from the present study reveal that a naturalistic mild stressor can enhance reconsolidation, improving the long-term expression of this declarative memory.
| Neuroscience. 2011 Apr 20. [Epub ahead of print]
The enhancement of reconsolidation with a naturalistic mild stressor improves the expression of a declarative memory in humans. Coccoz V, Maldonado H, Delorenzi A. Source Laboratorio de Neurobiología de la Memoria, Departamento de Fisiología y Biología Molecular y Celular, FCEyN, Universidad de Buenos Aires, IFIBYNE-CONICET, Argentina. Abstract The reconsolidation hypothesis proposes that a previously consolidated memory recalled by a reminder enters an unstable state (memory labilization) during which it is transiently sensitive to disruption. Although this process has been shown in very diverse species and types of memories, including human declarative memory, elucidating the role of this process is still an open challenge. The hypothesis that reconsolidation allows the incorporation of new information has recently been demonstrated in humans. However, the findings show that, during the reconsolidation phase, memory retention can be increased by pharmacological modulation or real live events in animals have not been found in humans yet. In order to evaluate this, we used a paradigm of human declarative memory whose reminder structure allows us to differentiate between a retrieved labile memory state and a retrieved but non-labile state. Volunteers learned an association between five cue-syllables and their respective response-syllables. 6 days later, the paired-associate memory was reactivated by exposing the subjects to the reminder, and then they received a mild stressor, cold pressor stress (CPS). Poor memory performance was found at both the time of memory reactivation (day 6 after training) and at testing of all groups that were designed as controls (day 7). Conversely, robust memory performance was shown at testing when the CPS administration was concurrent with the retrieved-labile memory state. Results from the present study reveal that a naturalistic mild stressor can enhance reconsolidation, improving the long-term expression of this declarative memory. This finding might have significant implications for the comprehension of memory persistence and memory expression, and add new evidence in order to understand the adaptive meaning of the reconsolidation process. Copyright © 2011. Published by Elsevier Ltd. |
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